Search results for "Adrenergic alpha-Antagonists"

showing 10 items of 22 documents

The 5-HT and alpha-adrenoceptor antagonist effect of four benzylisoquinoline alkaloids on rat aorta.

1998

Abstract The action of four benzylisoquinoline alkaloids (two aporphines—glaucine and apomorphine, a benzylisoquinoline—papaverine and a bisbenzyltetrahydroisoquinoline—antioquine) on 5-HT-induced contraction in rat thoracic aorta has been examined and compared with that of the control drugs: ketanserin, nifedipine, prazosin and phentolamine. The relaxant action on 5-HT-induced contraction was contrasted with that on the contraction induced by noradrenaline and KCl. The results obtained with control drugs show that ketanserin has clear selectivity for 5-HT receptors, whereas prazosin and phentolamine have high selectivity for the α1-adrenoceptor and nifedipine seems to have a more potent ef…

AgonistMalemedicine.medical_specialtySerotoninKetanserinAporphinesApomorphinemedicine.drug_classPharmaceutical ScienceAorta ThoracicIn Vitro TechniquesBenzylisoquinolinesMuscle Smooth Vascularchemistry.chemical_compoundPhentolamineAlkaloidsInternal medicinePapaverinemedicinePrazosinAnimalsRats WistarBenzylisoquinolineAdrenergic alpha-AntagonistsPharmacologyPapaverineDose-Response Relationship DrugChemistryParasympatholyticsCalcium Channel BlockersIsoquinolinesGlaucineRatsApomorphineEndocrinologyDopamine Agonistsmedicine.drugMuscle ContractionThe Journal of pharmacy and pharmacology
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Desensitization of inhibitory prejunctional alpha 2-adrenoceptors and putative imidazoline receptors on rabbit heart sympathetic nerves.

1993

To find out whether sympathetic nerves of the rabbit heart possess pharmacologically relevant prejunctional imidazoline receptors different from α-autoreceptors, the inhibition by oxymetazoline, aganodine and BDF 6143 (4-chloro-2-[2-imidazoline-2-ylamino]-isoindoline hydrochloride) of endogenous noradrenaline overflow evoked by stimulation of extrinsic postganglionic sympathetic nerves (0.66 Hz, 80 pulses) was investigated. In addition we wanted to find out whether either type of these prejunctional receptors undergoes desensitization upon pre-exposure to respective agonists. The α2-adrenoceptor agonist oxymetazoline inhibited the evoked noradrenaline overflow (2.9 nmol/l, IC50; about 90010…

Agonistmedicine.medical_specialtySympathetic Nervous Systemmedicine.drug_classReceptors DrugRauwolscineOxymetazolineImidazoline receptorStimulationMuscarinic agonistchemistry.chemical_compoundNorepinephrineReceptors Adrenergic alpha-2Internal medicinemedicinePrazosinAnimalsAdrenergic alpha-AntagonistsAutoreceptorsPharmacologyHeartGeneral MedicineEndocrinologychemistryAutoreceptorImidazoline ReceptorsRabbitsAdrenergic alpha-Agonistsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Functional evidence of inverse agonism in vascular smooth muscle

1996

1. In the present study, depletion of internal Ca2+ stores sensitive to noradrenaline (1 microM) in rat aorta, is the signal for the entry of extracellular Ca2+, not only to refill the stores but also, in our experimental conditions, to activate the contractile proteins. This induces an increase in the resting tone that constitutes, the first functional evidence of this Ca2+ entry. 2. The fact that methoxamine (100 microM) reproduces the same processes as noradrenaline but clonidine (1 microM) does not, indicates that alpha(1)-adrenoceptor activation is related to the increase in the resting tone observed after depletion of adrenoceptor-sensitive internal Ca2+-stores. 3. Benoxathian and WB …

Agonistmedicine.medical_specialtyVascular smooth musclemedicine.drug_classAlpha (ethology)Aorta ThoracicMuscle Smooth VascularMethoxamineDioxanesOxathiinsRats Sprague-DawleyNorepinephrinechemistry.chemical_compoundChloroethylclonidineInternal medicinemedicineAnimalsInverse agonistBenoxathianAdrenergic alpha-AntagonistsPharmacologyCell MembraneRatsEndocrinologychemistryAdrenergic alpha-1 Receptor AntagonistsCalciumAdrenergic alpha-1 Receptor Agonistsmedicine.symptomResearch ArticleMuscle ContractionMuscle contractionmedicine.drugBritish Journal of Pharmacology
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A possible structural determinant of selectivity of boldine and derivatives for the alpha 1A-adrenoceptor subtype.

1996

1. The selectivity of action of boldine and the related aporphine alkaloids, predicentrine (9-O-methylboldine) and glaucine (2,9-O-dimethylboldine) and alpha 1-adrenoceptor subtypes was studied by examining [3H]-prazosin competition binding in rat cerebral cortex. WB 4101 and benoxathian were used as selective alpha 1A-adrenoceptor antagonists. 2. In the competition experiments [3H]-prazosin (0.2 nM) binding was inhibited by WB 4101 and benoxathian. The inhibition curves displayed shallow slopes which could be subdivided into high and low affinity components (pKi = 9.92 and 8.29 for WB 4101, 9.35 and 7.94 for benoxathian). The two antagonists recognized approximately 37% of the sites with h…

AporphinesStereochemistryAlpha (ethology)Binding CompetitiveAntioxidantsDioxanesOxathiinschemistry.chemical_compoundChloroethylclonidineBoldineAnimalsAporphineBinding siteRats WistarBenoxathianAdrenergic alpha-AntagonistsPharmacologyCerebral CortexAlkaloidPrazosinGlaucineRatschemistryAdrenergic alpha-1 Receptor AntagonistsFemaleResearch Article
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Endotoxin inhibits gastric emptying in rats via a capsaicin-sensitive afferent pathway.

2001

The effects of endotoxin on gastric emptying of a solid nutrient meal and the neural mechanisms involved in such a response were investigated in conscious rats. The intraperitoneal (i.p.) administration of E. coli endotoxin (40 mug/kg) significantly reduced the 4-h rate of gastric emptying of a standard solid nutrient meal. Ablation of primary afferent neurons by systemic administration of high doses of capsaicin (20+30+50 mg/kg s.c.) to adult rats did not modify the rate of gastric emptying in control animals but prevented the delay in gastric transit induced by endotoxin. Local application of capsaicin to the vagus nerve rather than application of capsaicin to the celiac ganglion signific…

AtropineLipopolysaccharidesMaleendotoxinmedicine.medical_specialtyCalcitonin Gene-Related PeptidePharmacology toxicologyMuscarinic AntagonistscapsaicinRats Sprague-Dawleychemistry.chemical_compoundgastric emptyingtransitNeurons EfferentCalcitonin Gene-Related Peptide Receptor AntagonistsInternal medicinemedicineAnimalsDrug InteractionsCGRPNeurons AfferenttachykininsPhentolamineAfferent PathwayAdrenergic alpha-AntagonistsPharmacologyMealAfferent PathwaysGastric emptyingdigestive oral and skin physiologyGeneral MedicinePeptide FragmentsRatsEndocrinologychemistryGastric EmptyingCapsaicinCapsaicinNaunyn-Schmiedeberg's archives of pharmacology
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3-D image analysis of fluorescent drug binding

2004

Fluorescent ligands provide the means of studying receptors in whole tissues using confocal laser scanning microscopy and have advantages over antibody- or non-fluorescence-based method. Confocal microscopy provides large volumes of images to be measured. Histogram analysis of 3-D image volumes is proposed as a method of graphically displaying large amounts of volumetric image data to be quickly analyzed and compared. The fluorescent ligand BODIPY FL-prazosin (QAPB) was used in mouse aorta. Histogram analysis reports the amount of ligand-receptor binding under different conditions and the technique is sensitive enough to detect changes in receptor availability after antagonist incubation or…

Boron CompoundsMalelcsh:Medical technologyAdrenergic receptorBiomedical EngineeringMouse aortaIn Vitro TechniquesAntibodieslaw.inventionMiceImaging Three-DimensionalConfocal microscopylawReceptors Adrenergic alpha-1HistogramAnimalsRadiology Nuclear Medicine and imagingReceptorlcsh:QH301-705.5Adrenergic alpha-AntagonistsAortaFluorescent DyesMice KnockoutMicroscopy ConfocalDose-Response Relationship DrugPhenoxybenzamineChemistryPrazosinBiological tissueCondensed Matter PhysicsFluorescenceAutofluorescencelcsh:Biology (General)lcsh:R855-855.5BiophysicsMolecular MedicineFemaleBiotechnology
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On the mechanism of action of phenylephrine in rat atrial heart muscle

1994

Both in rat left atrial heart and in aortic smooth muscle preparations, phenylephrine (PE) caused a concentration-dependent increase in force of contraction (FC) in the presence of atenolol (10 mumol/l), which was antagonized by phentolamine, prazosin and WB 4101 in a competitive manner. The pA2 values of the antagonists in the cardiac tissue were 10-20fold lower than those in the rat thoracic aorta. In the spontaneously beating right atrium, PE exerted a positive chronotropic action, which was not significantly antagonized by phentolamine or prazosin. It is therefore assumed that the effects of phenylephrine in the left atrium and in the aorta are mediated by different subtypes of alpha 1-…

MaleChronotropicmedicine.medical_specialtyPotassium ChannelsSodium-Hydrogen ExchangersAction PotentialsIn Vitro TechniquesRats Sprague-DawleyPhenylephrinePhentolamineHeart RateReceptors Adrenergic alpha-1medicine.arteryInternal medicinemedicinePrazosinAnimalsHeart AtriaPhenylephrineAdrenergic alpha-AntagonistsPharmacologyAortaChemistryCalcium RadioisotopesHeartGeneral MedicineAtenololMyocardial ContractionRatsElectrophysiologyActin CytoskeletonEndocrinologyMechanism of actioncardiovascular systemCalciummedicine.symptomAdrenergic alpha-Agonistsmedicine.drugMuscle contractionNaunyn-Schmiedeberg’s Archives of Pharmacology
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Effects of prostaglandin E1 and buflomedil on left ventricular function in patients with severe chronic occlusive arterial disease: a prospective, ra…

1999

In this study, the effect of a course of prostaglandin E ( 1 ) (60 microg/d intravenously [i.v.]) or buflomedil (150 mg/d i.v.) treatment on parameters of left ventricular systolic function was investigated by echocardiography in patients of comparatively advanced age with severe peripheral occlusive arterial disease (Fontaine's stage III or IV). The study population was 20 patients, 12 men and 8 women, between 51 and 85 years of age (average age, 73. 7 years), with multiple coexisting medical conditions. These patients were no longer suitable candidates for other forms of interventional or surgical treatment. The patients were treated with prostaglandin E ( 1 ) or buflomedil in the dosages…

MalePyrrolidinesDosemedicine.medical_treatmentLeft Ventricular Ejection TimeArterial Occlusive DiseasesVentricular Function Leftchemistry.chemical_compoundDouble-Blind MethodBuflomedilMedicineHumansPharmacology (medical)Prospective StudiesAlprostadilProstaglandin E1Adrenergic alpha-AntagonistsAgedPharmacologyAged 80 and overPeripheral Vascular DiseasesEjection fractionbusiness.industryGeneral MedicineMiddle AgedPeripheralchemistryAnesthesiaChronic DiseasePopulation studyFemalebusinessProstaglandin EAmerican journal of therapeutics
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Adrenergic modulation of the release of 5-hydroxytryptamine from the vascularly perfused ileum of the guinea-pig

1988

1. Isolated segments of the guinea-pig ileum were vascularly perfused and the release of 5-hydroxytryptamine (5-HT) and 5-hydroxyindoleacetic acid (5-HIAA) into the portal venous effluent was determined by h.p.l.c. with electrochemical detection. Test substances were applied via the arterial perfusion medium. 2. Isoprenaline (0.1 microM) increased the outflow of 5-HT and 5-HIAA maximally by about 75% and this was antagonized by propranolol (0.1 microM). Forskolin (1-10 microM) increased the outflow of 5-HT by approximately 105% and that of 5-HIAA by approximately 55%. The phosphodiesterase inhibitor AH 21-132 (0.1-1 microM) increased the outflow of 5-HT and 5-HIAA by about 70%. Isoprenaline…

MaleSerotoninmedicine.medical_specialtyPhosphodiesterase InhibitorsAdrenergic beta-AntagonistsGuinea PigsPropranololClonidinechemistry.chemical_compoundIleumIsoprenalineInternal medicinemedicinePrazosinAnimalsNaphthyridinesPhosphodiesterase inhibitorAdrenergic alpha-AntagonistsPharmacologyForskolinColforsinIsoproterenolPhosphodiesteraseAdrenergic beta-AgonistsHydroxyindoleacetic AcidPerfusionEndocrinologychemistryTetrodotoxinEnterochromaffin cellAdrenergic alpha-AgonistsResearch Articlemedicine.drugBritish Journal of Pharmacology
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The effect of S-(+)-boldine on the α1-adrenoceptor of the guinea-pig aorta

1996

1. The cardiovascular activity of S-(+)-boldine, an aporphine alkaloid structurally related to papaverine, was determined. The work includes functional studies on guinea-pig isolated aorta contracted with noradrenaline, caffeine, KCl or Ca2+, and on guinea-pig trachea contracted with acetylcholine or histamine. 2. S-(+)-boldine inhibited in a concentration-dependent manner the contractile response evoked by noradrenaline (10 microM) in guinea-pig aorta (IC50 = 1.4 +/- 0.2 microM) while the KCl depolarizing solution (60 mM)- or the Ca2+ (1 mM)-induced contractions were only partially affected by boldine up to 300 microM. In contrast, papaverine relaxed noradrenaline (NA), KCl or Ca2+ induced…

Malemedicine.medical_specialtyAporphinesContraction (grammar)Phosphodiesterase InhibitorsMuscle RelaxationGuinea PigsAorta ThoracicIn Vitro TechniquesPhosphatidylinositolsMuscle Smooth Vascularchemistry.chemical_compoundCaffeinePapaverineReceptors Adrenergic alpha-1Internal medicinemedicine.arterymedicinePrazosinAnimalsBoldineThoracic aortaAdrenergic alpha-AntagonistsPharmacologyPapaverineParasympatholyticsCalcium Channel BlockersTracheaEndocrinologychemistryCompetitive antagonistAdrenergic alpha-1 Receptor AntagonistsHistamineAcetylcholineResearch Articlemedicine.drugBritish Journal of Pharmacology
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